COVID modRNA Transfections form Spike Product Clumps in Heart Cells

An in vitro study that investigated the intracellular behavior of COVID modRNA transfections in three types of cultured cell lines (Human AC16 cardiomyocytes, HEK-293 cells and HeLa cells) found that both the Moderna and Pfizer modRNA transfections formed clumps of spike monomers and subunits within each cell type. In cardiomyocytes these clumps impaired cell proliferation, induced pro-inflammatory responses, and caused oxidative stress. The authors suggest that the generation of these off-target, aggregated spike products may explain some acute and long-term cardiac adverse events following the COVID vaxx particularly modRNA.

A case control study consisting of participants with post vaxx or post infection syndrome symptoms (and healthy controls) who received the Pfizer modRNA transfections in 2021-22 (n = 30) found, after collecting blood samples and conducting a protein analysis, that both the post vaxx and post infection syndrome cohorts had a significant increase in coagulation proteins factors 10 and 11 along with an increase in acute inflammatory response proteins which evidences the formation of microclots caused by misfolding proteins that formed amyloid-type fibrils.

As I mentioned last year in a prior post, a study involving 3 experiments conducted with rodents, human cell culture and rodent Langendorff hearts modRNA lipids and translated spike protein were demonstrated to be a cardiotoxin that cause stress induced cardiomyopathy. An autopsy study of two teenage boys who died shortly after receiving their second modRNA doses supported these findings.